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Showing 1 to 12 of 335 entries
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Expression of concern.

Clinical and experimental immunology

[No authors listed]
PMID: 24494672
Clin Exp Immunol. 2014 Mar;175(3):498. doi: 10.1111/cei.12257.

No abstract available.

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Expression of concern. Clin Exp Immunol. 2014;175(3):498doi: 10.1111/cei.12257.
(2014). Expression of concern. Clinical and experimental immunology, 175(3), 498. https://doi.org/10.1111/cei.12257
"Expression of concern." Clinical and experimental immunology vol. 175,3 (2014): 498. doi: https://doi.org/10.1111/cei.12257
Expression of concern. Clin Exp Immunol. 2014 Mar;175(3):498. doi: 10.1111/cei.12257. PMID: 24494672; PMCID: PMC3927910.
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Reversal of oxidative stress-induced apoptosis in T and B lymphocytes by Coenzyme Q10 (CoQ10).

American journal of clinical and experimental immunology

Gollapudi S, Gupta S.
PMID: 27168954
Am J Clin Exp Immunol. 2016 Apr 27;5(2):41-7. eCollection 2016.

Coenzyme Q10, (CoQ10) an electron transporter and an antioxidant, protects a variety of cell types against oxidative stress and apoptosis. However, protective effect of CoQ10 on oxidative stress-induced apoptosis in lymphocytes has not been studied in detail. In this...

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Gollapudi S, Gupta S. Reversal of oxidative stress-induced apoptosis in T and B lymphocytes by Coenzyme Q10 (CoQ10). Am J Clin Exp Immunol. 2016;5(2):41-7
Gollapudi, S., & Gupta, S. (2016). Reversal of oxidative stress-induced apoptosis in T and B lymphocytes by Coenzyme Q10 (CoQ10). American journal of clinical and experimental immunology, 5(2), 41-7.
Gollapudi, Sastry, and Gupta, Sudhir. "Reversal of oxidative stress-induced apoptosis in T and B lymphocytes by Coenzyme Q10 (CoQ10)." American journal of clinical and experimental immunology vol. 5,2 (2016): 41-7.
Gollapudi S, Gupta S. Reversal of oxidative stress-induced apoptosis in T and B lymphocytes by Coenzyme Q10 (CoQ10). Am J Clin Exp Immunol. 2016 Apr 27;5(2):41-7. eCollection 2016. PMID: 27168954; PMCID: PMC4858601.
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Analysis of subsets of B cells, Breg, CD4Treg and CD8Treg cells in adult patients with primary selective IgM deficiency.

American journal of clinical and experimental immunology

Louis AG, Agrawal S, Gupta S.
PMID: 27168952
Am J Clin Exp Immunol. 2016 Mar 23;5(1):21-32. eCollection 2016.

Primary selective IgM deficiency (SIGMD) is a rare and recently IUIS-recognized primary immunodeficiency disease with increased susceptibility to infections, allergy, and autoimmune diseases. The pathogenesis of selective IgM remains unclear. The objective of the study was to understand the...

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Louis AG, Agrawal S, Gupta S. Analysis of subsets of B cells, Breg, CD4Treg and CD8Treg cells in adult patients with primary selective IgM deficiency. Am J Clin Exp Immunol. 2016;5(1):21-32
Louis, A. G., Agrawal, S., & Gupta, S. (2016). Analysis of subsets of B cells, Breg, CD4Treg and CD8Treg cells in adult patients with primary selective IgM deficiency. American journal of clinical and experimental immunology, 5(1), 21-32.
Louis, Ankmalika Gupta, et al. "Analysis of subsets of B cells, Breg, CD4Treg and CD8Treg cells in adult patients with primary selective IgM deficiency." American journal of clinical and experimental immunology vol. 5,1 (2016): 21-32.
Louis AG, Agrawal S, Gupta S. Analysis of subsets of B cells, Breg, CD4Treg and CD8Treg cells in adult patients with primary selective IgM deficiency. Am J Clin Exp Immunol. 2016 Mar 23;5(1):21-32. eCollection 2016. PMID: 27168952; PMCID: PMC4858603.
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Glycyrrhizin down-regulates CCL2 and CXCL2 expression in cerulein-stimulated pancreatic acinar cells.

American journal of clinical and experimental immunology

Panahi Y, Fakhari S, Mohammadi M, Rahmani MR, Hakhamaneshi MS, Jalili A.
PMID: 26155433
Am J Clin Exp Immunol. 2015 Jul 05;4(1):1-6. eCollection 2015.

Many inflammatory chemokines release from leukocytes and pancreatic acinar cells which play important roles in pathophysiology of acute pancreatitis (AP). Of interests, CXCL2 and CCL2 have been shown elevated in the plasma of patients with AP. We have recently...

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Panahi Y, Fakhari S, Mohammadi M, et al. Glycyrrhizin down-regulates CCL2 and CXCL2 expression in cerulein-stimulated pancreatic acinar cells. Am J Clin Exp Immunol. 2015;4(1):1-6
Panahi, Y., Fakhari, S., Mohammadi, M., Rahmani, M. R., Hakhamaneshi, M. S., & Jalili, A. (2015). Glycyrrhizin down-regulates CCL2 and CXCL2 expression in cerulein-stimulated pancreatic acinar cells. American journal of clinical and experimental immunology, 4(1), 1-6.
Panahi, Yaser, et al. "Glycyrrhizin down-regulates CCL2 and CXCL2 expression in cerulein-stimulated pancreatic acinar cells." American journal of clinical and experimental immunology vol. 4,1 (2015): 1-6.
Panahi Y, Fakhari S, Mohammadi M, Rahmani MR, Hakhamaneshi MS, Jalili A. Glycyrrhizin down-regulates CCL2 and CXCL2 expression in cerulein-stimulated pancreatic acinar cells. Am J Clin Exp Immunol. 2015 Jul 05;4(1):1-6. eCollection 2015. PMID: 26155433; PMCID: PMC4494114.
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Investigation of IL-21 gene polymorphisms (rs2221903, rs2055979) in cases with multiple sclerosis of Azerbaijan, Northwest Iran.

American journal of clinical and experimental immunology

Gharibi T, Kazemi T, Aliparasti MR, Farhoudi M, Almasi S, Dehghanzadeh R, Seyfizadeh N, Babaloo Z.
PMID: 26155434
Am J Clin Exp Immunol. 2015 Jul 05;4(1):7-14. eCollection 2015.

BACKGROUND: Multiple Sclerosis (MS) is a chronic inflammatory demyelinating disease of the Central Nervous System that is immunologically mediated in genetically susceptible individuals. IL-21, a cytokine produced by TCD4(+) cells, particularly by Th-17 cells, is believed to play an...

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Gharibi T, Kazemi T, Aliparasti MR, et al. Investigation of IL-21 gene polymorphisms (rs2221903, rs2055979) in cases with multiple sclerosis of Azerbaijan, Northwest Iran. Am J Clin Exp Immunol. 2015;4(1):7-14
Gharibi, T., Kazemi, T., Aliparasti, M. R., Farhoudi, M., Almasi, S., Dehghanzadeh, R., Seyfizadeh, N., & Babaloo, Z. (2015). Investigation of IL-21 gene polymorphisms (rs2221903, rs2055979) in cases with multiple sclerosis of Azerbaijan, Northwest Iran. American journal of clinical and experimental immunology, 4(1), 7-14.
Gharibi, Tohid, et al. "Investigation of IL-21 gene polymorphisms (rs2221903, rs2055979) in cases with multiple sclerosis of Azerbaijan, Northwest Iran." American journal of clinical and experimental immunology vol. 4,1 (2015): 7-14.
Gharibi T, Kazemi T, Aliparasti MR, Farhoudi M, Almasi S, Dehghanzadeh R, Seyfizadeh N, Babaloo Z. Investigation of IL-21 gene polymorphisms (rs2221903, rs2055979) in cases with multiple sclerosis of Azerbaijan, Northwest Iran. Am J Clin Exp Immunol. 2015 Jul 05;4(1):7-14. eCollection 2015. PMID: 26155434; PMCID: PMC4494115.
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Retraction.

Clinical and experimental immunology

Minakawa Y, Peakman M.
PMID: 19058349
Clin Exp Immunol. 2008 Dec;154(3):433-4. doi: 10.1111/j.1365-2249.2008.03824.x.

No abstract available.

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Minakawa Y, Peakman M. Retraction. Clin Exp Immunol. 2008;154(3):433-4doi: 10.1111/j.1365-2249.2008.03824.x.
Minakawa, Y., & Peakman, M. (2008). Retraction. Clinical and experimental immunology, 154(3), 433-4. https://doi.org/10.1111/j.1365-2249.2008.03824.x
Minakawa, Youichi, and Peakman, M. "Retraction." Clinical and experimental immunology vol. 154,3 (2008): 433-4. doi: https://doi.org/10.1111/j.1365-2249.2008.03824.x
Minakawa Y, Peakman M. Retraction. Clin Exp Immunol. 2008 Dec;154(3):433-4. doi: 10.1111/j.1365-2249.2008.03824.x. PMID: 19058349; PMCID: PMC2633241.
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Inter-donor variation in cell subset specific immune signaling responses in healthy individuals.

American journal of clinical and experimental immunology

Longo DM, Louie B, Wang E, Pos Z, Marincola FM, Hawtin RE, Cesano A.
PMID: 23885311
Am J Clin Exp Immunol. 2012 Apr 24;1(1):1-11. Print 2012.

Single cell network profiling (SCNP) is a multi-parameter flow cytometry based approach that allows for the simultaneous interrogation of intracellular signaling pathways in multiple cell subpopulations within heterogeneous tissues, without the need for individual cell subset isolation. Thus, the...

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Longo DM, Louie B, Wang E, et al. Inter-donor variation in cell subset specific immune signaling responses in healthy individuals. Am J Clin Exp Immunol. 2012;1(1):1-11
Longo, D. M., Louie, B., Wang, E., Pos, Z., Marincola, F. M., Hawtin, R. E., & Cesano, A. (2012). Inter-donor variation in cell subset specific immune signaling responses in healthy individuals. American journal of clinical and experimental immunology, 1(1), 1-11.
Longo, Diane M, et al. "Inter-donor variation in cell subset specific immune signaling responses in healthy individuals." American journal of clinical and experimental immunology vol. 1,1 (2012): 1-11.
Longo DM, Louie B, Wang E, Pos Z, Marincola FM, Hawtin RE, Cesano A. Inter-donor variation in cell subset specific immune signaling responses in healthy individuals. Am J Clin Exp Immunol. 2012 Apr 24;1(1):1-11. Print 2012. PMID: 23885311; PMCID: PMC3714187.
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Gastrointestinal-associated autoantibodies in different autoimmune diseases.

American journal of clinical and experimental immunology

Shor DB, Orbach H, Boaz M, Altman A, Anaya JM, Bizzaro N, Tincani A, Cervera R, Espinosa G, Stojanovich L, Rozman B, Bombardieri S, Vita SD, Damoiseaux J, Villalta D, Tonutti E, Tozzoli R, Barzilai O, Ram M, Blank M, Agmon-Levin N, Shoenfeld Y.
PMID: 23885314
Am J Clin Exp Immunol. 2012 May 25;1(1):49-55. Print 2012.

BACKGROUND: Gastrointestinal (GI)-related autoantibodies (Abs) are rarely evaluated in autoimmune diseases (AID) other than inflammatory bowel disease, autoimmune hepatitis and celiac disease. Our aim was to determine the prevalence of these antibodies in a wide spectrum of AID.METHODS: We...

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Shor DB, Orbach H, Boaz M, et al. Gastrointestinal-associated autoantibodies in different autoimmune diseases. Am J Clin Exp Immunol. 2012;1(1):49-55
Shor, D. B., Orbach, H., Boaz, M., Altman, A., Anaya, J. M., Bizzaro, N., Tincani, A., Cervera, R., Espinosa, G., Stojanovich, L., Rozman, B., Bombardieri, S., Vita, S. D., Damoiseaux, J., Villalta, D., Tonutti, E., Tozzoli, R., Barzilai, O., Ram, M., Blank, M., Agmon-Levin, N., & Shoenfeld, Y. (2012). Gastrointestinal-associated autoantibodies in different autoimmune diseases. American journal of clinical and experimental immunology, 1(1), 49-55.
Shor, Dana Ben-Ami, et al. "Gastrointestinal-associated autoantibodies in different autoimmune diseases." American journal of clinical and experimental immunology vol. 1,1 (2012): 49-55.
Shor DB, Orbach H, Boaz M, Altman A, Anaya JM, Bizzaro N, Tincani A, Cervera R, Espinosa G, Stojanovich L, Rozman B, Bombardieri S, Vita SD, Damoiseaux J, Villalta D, Tonutti E, Tozzoli R, Barzilai O, Ram M, Blank M, Agmon-Levin N, Shoenfeld Y. Gastrointestinal-associated autoantibodies in different autoimmune diseases. Am J Clin Exp Immunol. 2012 May 25;1(1):49-55. Print 2012. PMID: 23885314; PMCID: PMC3714189.
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Potent suppression of arginase 1 expression in murine macrophages by low dose endotoxin.

American journal of clinical and experimental immunology

Surace MJ, Li L.
PMID: 23885329
Am J Clin Exp Immunol. 2013 Feb 27;2(1):117-23. Print 2013.

Macrophages can respond to diverse signals and adopt multiple phenotypes. Although interleukin-4 (IL-4) is shown to potently induce the expression of arginase 1 and contribute to differentiation of macrophages to the anti-inflammatory M2 phenotype, other modulators may potentiate or...

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Surace MJ, Li L. Potent suppression of arginase 1 expression in murine macrophages by low dose endotoxin. Am J Clin Exp Immunol. 2013;2(1):117-23
Surace, M. J., & Li, L. (2013). Potent suppression of arginase 1 expression in murine macrophages by low dose endotoxin. American journal of clinical and experimental immunology, 2(1), 117-23.
Surace, Michael J, and Li, Liwu. "Potent suppression of arginase 1 expression in murine macrophages by low dose endotoxin." American journal of clinical and experimental immunology vol. 2,1 (2013): 117-23.
Surace MJ, Li L. Potent suppression of arginase 1 expression in murine macrophages by low dose endotoxin. Am J Clin Exp Immunol. 2013 Feb 27;2(1):117-23. Print 2013. PMID: 23885329; PMCID: PMC3714206.
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Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology.

American journal of clinical and experimental immunology

Bourigault ML, Vacher R, Rose S, Olleros ML, Janssens JP, Quesniaux VF, Garcia I.
PMID: 23885330
Am J Clin Exp Immunol. 2013 Feb 27;2(1):124-34. Print 2013.

Tuberculosis (TB) is a major health problem requiring sustained immunity to inhibit Mycobacterium tuberculosis growth and appropriate antimicrobial therapy to prevent dissemination and drug resistance. Cell-mediated immune responses to M. tuberculosis involve the activation of cytokines such as Tumor...

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Bourigault ML, Vacher R, Rose S, et al. Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology. Am J Clin Exp Immunol. 2013;2(1):124-34
Bourigault, M. L., Vacher, R., Rose, S., Olleros, M. L., Janssens, J. P., Quesniaux, V. F., & Garcia, I. (2013). Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology. American journal of clinical and experimental immunology, 2(1), 124-34.
Bourigault, Marie-Laure, et al. "Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology." American journal of clinical and experimental immunology vol. 2,1 (2013): 124-34.
Bourigault ML, Vacher R, Rose S, Olleros ML, Janssens JP, Quesniaux VF, Garcia I. Tumor necrosis factor neutralization combined with chemotherapy enhances Mycobacterium tuberculosis clearance and reduces lung pathology. Am J Clin Exp Immunol. 2013 Feb 27;2(1):124-34. Print 2013. PMID: 23885330; PMCID: PMC3714199.
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Shaping the (auto)immune response in the gut: the role of intestinal immune regulation in the prevention of type 1 diabetes.

American journal of clinical and experimental immunology

Sorini C, Falcone M.
PMID: 23885333
Am J Clin Exp Immunol. 2013 Jun 15;2(2):156-71. Print 2013.

The pathogenesis of organ-specific autoimmune diseases such as Type 1 Diabetes (T1D) is regulated by genetic and environmental factors. There is increasing evidence that environmental factors acting at the intestinal level, with a special regard to the diverse bacterial...

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Sorini C, Falcone M. Shaping the (auto)immune response in the gut: the role of intestinal immune regulation in the prevention of type 1 diabetes. Am J Clin Exp Immunol. 2013;2(2):156-71
Sorini, C., & Falcone, M. (2013). Shaping the (auto)immune response in the gut: the role of intestinal immune regulation in the prevention of type 1 diabetes. American journal of clinical and experimental immunology, 2(2), 156-71.
Sorini, Chiara, and Falcone, Marika. "Shaping the (auto)immune response in the gut: the role of intestinal immune regulation in the prevention of type 1 diabetes." American journal of clinical and experimental immunology vol. 2,2 (2013): 156-71.
Sorini C, Falcone M. Shaping the (auto)immune response in the gut: the role of intestinal immune regulation in the prevention of type 1 diabetes. Am J Clin Exp Immunol. 2013 Jun 15;2(2):156-71. Print 2013. PMID: 23885333; PMCID: PMC3714176.
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SOCS proteins in development and disease.

American journal of clinical and experimental immunology

Trengove MC, Ward AC.
PMID: 23885323
Am J Clin Exp Immunol. 2013 Feb 27;2(1):1-29. Print 2013.

Cytokine and growth factor signaling mediates essential roles in the differentiation, proliferation, survival and function of a number of cell lineages. This is achieved via specific receptors located on the surface of target cells, with ligand binding activating key...

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Trengove MC, Ward AC. SOCS proteins in development and disease. Am J Clin Exp Immunol. 2013;2(1):1-29
Trengove, M. C., & Ward, A. C. (2013). SOCS proteins in development and disease. American journal of clinical and experimental immunology, 2(1), 1-29.
Trengove, Monique C, and Ward, Alister C. "SOCS proteins in development and disease." American journal of clinical and experimental immunology vol. 2,1 (2013): 1-29.
Trengove MC, Ward AC. SOCS proteins in development and disease. Am J Clin Exp Immunol. 2013 Feb 27;2(1):1-29. Print 2013. PMID: 23885323; PMCID: PMC3714205.
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Showing 1 to 12 of 335 entries